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GC−MS analysis of 786-O and isogenic SETD2-deficient 38E/38F

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Fatty Acids Analysis Using Gas Chromatography-Mass Spectrometer Detector (GC/MSD) - Method Validation Based on Berry Seed Extract Samples

Integrated Genomic and Proteomic Analyses Reveal Novel Mechanisms of the Methyltransferase SETD2 in Renal Cell Carcinoma Development - ScienceDirect

PDF) Loss of SETD2 Induces a Metabolic Switch in Renal Cell Carcinoma Cell Lines toward Enhanced Oxidative Phosphorylation

SETD2 deficiency accelerates sphingomyelin accumulation and promotes the development of renal cancer

PDF) Loss of SETD2 Induces a Metabolic Switch in Renal Cell Carcinoma Cell Lines toward Enhanced Oxidative Phosphorylation

Integrated Genomic and Proteomic Analyses Reveal Novel Mechanisms of the Methyltransferase SETD2 in Renal Cell Carcinoma Development - ScienceDirect

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SETD2 loss-of-function promotes renal cancer branched evolution through replication stress and impaired DNA repair

SETD2 deficiency accelerates sphingomyelin accumulation and promotes the development of renal cancer

Applications of Mass Spectrometric Techniques to the Analysis of Fuels and Lubricants

SETD2 loss-of-function promotes renal cancer branched evolution through replication stress and impaired DNA repair

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