GC−MS analysis of 786-O and isogenic SETD2-deficient 38E/38F
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Integrated Genomic and Proteomic Analyses Reveal Novel Mechanisms of the Methyltransferase SETD2 in Renal Cell Carcinoma Development - ScienceDirect
PDF) Loss of SETD2 Induces a Metabolic Switch in Renal Cell Carcinoma Cell Lines toward Enhanced Oxidative Phosphorylation
SETD2 deficiency accelerates sphingomyelin accumulation and promotes the development of renal cancer
PDF) Loss of SETD2 Induces a Metabolic Switch in Renal Cell Carcinoma Cell Lines toward Enhanced Oxidative Phosphorylation
Integrated Genomic and Proteomic Analyses Reveal Novel Mechanisms of the Methyltransferase SETD2 in Renal Cell Carcinoma Development - ScienceDirect
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SETD2 loss-of-function promotes renal cancer branched evolution through replication stress and impaired DNA repair
SETD2 deficiency accelerates sphingomyelin accumulation and promotes the development of renal cancer
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SETD2 loss-of-function promotes renal cancer branched evolution through replication stress and impaired DNA repair
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